Schiffs Diseases of the Liver (2 Volume Set) 10th ed

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Home Contact us Help Free delivery worldwide. Free delivery worldwide. Bestselling Series. Harry Potter. Popular Features. New Releases. Notify me. Description The premier clinical hepatology reference for 50 years, Schiff's Diseases of the Liver is in its Tenth Edition--now in full color throughout. This edition features a major new transplant section focusing on pre-transplant and post-transplant evaluation and management.

Also included are updates on key topics such as hepatitis, NASH, and drug-induced pathologies. The first third of the book covers anatomy, pathology, testing, imaging, and effects of liver disease on other organs. Subsequent sections address specific diseases and clinical syndromes.

Schiff's Diseases Of The Liver. 2 Volume Set

Each of the 12 sections begins with an overview, and each chapter starts with an outline of key concepts. Numerous clinical algorithms appear throughout the text. Product details Format Hardback pages Dimensions x x On examination, the child had hepatosplenomegaly with jaundice, while the other systems were normal.

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Investigations showed hemoglobin of Bilirubin was 5. Prothrombin time and partial thromboplastin time were normal. Urine reducing substance was negative and ultrasound abdomen showed hepatosplenomegaly and presence of gall bladder. Hepatobiliary iminodiacetic acid scan HIDA scan was normal.

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Ultrasound of the skull showed mild prominence of both lateral ventricles. No other neuroimaging tests were done due to nonaffordability. Cerebrospinal fluid examination was not done and neurological examination was normal. The child had normal achievement of milestones during the year and jaundice resolved by 5 months of age.

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At the end of 1 year, her weight was 6. Her hearing and ophthalmology examination was also normal. If the neonates survive, they present with cerebral symptoms of convulsions, tremors, paralysis, impaired intellect and behavioral problems. Focal areas of necrosis may be seen in the viscera, leading to myocarditis and pneumonitis and, rarely, hepatitis.

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Callahan et al. Sabin suggested that the jaundice might be due to a direct toxic effect of Toxoplasma on the liver cells. In our patient though, we could not demonstrate Toxoplasma in the liver tissue, while rising Toxoplasma IgG titer was suggestive of infection and treatment led to resolution of hepatitis.

Thus, while determining the cause of neonatal hepatitis, congenital toxoplasmosis should be considered as a possibility, even when the accompanying constellation of symptoms is absent, and benefit of therapy should be given on confirmation of diagnosis.

Source of Support: Nil. Conflict of Interest: None declared. National Center for Biotechnology Information , U. Journal List J Clin Neonatol v. J Clin Neonatol. Author information Copyright and License information Disclaimer. Address for correspondence: Dr. This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3. Abstract Congenital toxoplasmosis may be subclinical, or may present with multisystem involvement. Keywords: Chorioretinitis, congenital, neonatal hepatitis, toxoplasmosis.

Schiff's Diseases of the Liver / Edition 10

Congenital Toxoplasmosis: Infections in Neonates. Desmonts G, Couvreur J. Congenital toxoplasmosis. Prospective study of the outcome of pregnancy in women with toxoplasmosis acquired during pregnancy. Ann Pediatr Paris ; 31 —9.

Rosenthal P. Neonatal hepatitis and congenital infections. Liver disease in children.